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Reuters: Pot Kills Cancer But Don’t Even Think About Using It!
Wed, 19 Aug 2009 04:25:44 By: Paul Armentano, NORML Deputy Director
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It was just yesterday that I was lamenting about the mainstream media’s failure to report on the anti-cancer properties of cannabis. And then along comes Reuters with this:
Cannabis chemicals may help fight prostate cancer
via Reuters News Wire
Chemicals in cannabis have been found to stop prostate cancer cells from growing in the laboratory, suggesting that cannabis-based medicines could one day help fight the disease, scientists said Wednesday.
After working initially with human cancer cell lines, Ines Diaz-Laviada and colleagues from the University of Alcala in Madrid also tested one compound on mice and discovered it produced a significant reduction in tumor growth.
Their research, published in the British Journal of Cancer, underlines the growing interest in the medical use of active chemicals called cannabinoids, which are found in marijuana.
Experts, however, stressed that the research was still exploratory and many more years of testing would be needed to work out how to apply the findings to the treatment of cancer in humans.
“This is interesting research which opens a new avenue to explore potential drug targets but it is at a very early stage,” said Lesley Walker, director of cancer information at Cancer Research UK, which owns the journal.
“It absolutely isn’t the case that men might be able to fight prostate cancer by smoking cannabis,” she added.
Well, well, well, leave it to the MSM to misrepresent the facts and miss the real story. First, the chemicals assessed in this study, R(+)-Methanandamide and JWH-015, are neither “cannabinoids” nor are they “chemicals in cannabis.” Rather, they are synthetic, selective CB2 receptor agonists. In short, they are chemicals created in a lab to mimic certain elements in marijuana, and to bind to specifically to those cannabinoid receptors that are not located in the brain. After all, we can’t possibly have the terminally ill feeling ‘better’, now can we?
Second, US federal researchers have known for some 35 years that the naturally occurring chemicals in cannabis — not just synthesized agonists — can halt the proliferation of multiple types of cancer, including including brain cancer, prostate cancer, breast cancer, lung cancer, skin cancer, and pancreatic cancer. We even know how.
Cannabinoids: potential anticancer agents
via Nature Reviews Cancer (2003)
Cannabinoids are usually well tolerated, and do not produce the generalized toxic effects of conventional chemotherapies. … Cannabinoids inhibit tumor growth in laboratory animals. They do so by modulating key cell-signaling pathways, thereby inducing direct growth arrest and death of tumor cells, as well as by inhibiting tumor angiogenesis and metastasis. Cannabinoids are selective anti-tumor compounds, as they can kill tumor cells without affecting their non-transformed counterparts.
Of course, the real question — conveniently ignored by Reuters and the rest of the MSM — is this: Why, after three decades and dozens of preclinical trials documenting cannabis’ potent anti-cancer abilities, are “many more years of testing” necessary? Last I checked, humans die en masse from cancer, not rats! Yet for some 35 years scientists have been content to replicate these cancer-killer findings in animals and in petri dishes, all the while warning, “It absolutely isn’t the case that men might be able to fight prostate cancer by smoking cannabis.”
Well why the hell not? Not only can cannabis alleviate cancer patients’ nausea and pain, elevate their mood, and increase their appetite, but also — as dozens of preclinical trials over the past three decades consistently demonstrate — marijuana may help to alleviate the very disease that’s ravaging their bodies. Of course, rather than put this theory to the test, investigators for more than three decades have been willing to let people with a terminal illness die while they piddle around with their petri dishes. And to date, not one reporter from the mainstream media has ever had the guts to ask them why.
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If Cannabis Smoking Didn’t Adversely Impact Lung Function You Would Have Read About It, Right?
Tue, 18 Aug 2009 20:51:23 By: Paul Armentano, NORML Deputy Director
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To follow up on yesterday’s blog post, here are the findings of yet another just published study that the mainstream media will undoubtedly ignore.
Effects of cannabis on lung function: a population-based cohort study
via nih.gov
The effects of cannabis on lung function remain unclear and may be different to tobacco. We compared the associations between use of these substances and lung function in a population-based cohort (n=1037). … Cumulative cannabis use was associated with higher forced vital capacity, total lung capacity, functional residual capacity, and residual volume. Cannabis was also associated with higher airways resistance but not with forced expiratory volume in 1 second, forced expiratory ratio, or transfer factor. These findings were similar amongst those who did not smoke tobacco.
By contrast, tobacco use was associated with lower forced expiratory volume in 1 second, lower forced expiratory ratio, lower transfer factor, and higher static lung volumes, but not with airways resistance. Cannabis appears to have different effects on lung function to those of tobacco.
Just in case you think that this is the first time that researchers have failed to document a decline in lung function in marijuana users, well, think again. And again. And again.
Wed, 19 Aug 2009 04:25:44 By: Paul Armentano, NORML Deputy Director
Share This Article
It was just yesterday that I was lamenting about the mainstream media’s failure to report on the anti-cancer properties of cannabis. And then along comes Reuters with this:
Cannabis chemicals may help fight prostate cancer
via Reuters News Wire
Chemicals in cannabis have been found to stop prostate cancer cells from growing in the laboratory, suggesting that cannabis-based medicines could one day help fight the disease, scientists said Wednesday.
After working initially with human cancer cell lines, Ines Diaz-Laviada and colleagues from the University of Alcala in Madrid also tested one compound on mice and discovered it produced a significant reduction in tumor growth.
Their research, published in the British Journal of Cancer, underlines the growing interest in the medical use of active chemicals called cannabinoids, which are found in marijuana.
Experts, however, stressed that the research was still exploratory and many more years of testing would be needed to work out how to apply the findings to the treatment of cancer in humans.
“This is interesting research which opens a new avenue to explore potential drug targets but it is at a very early stage,” said Lesley Walker, director of cancer information at Cancer Research UK, which owns the journal.
“It absolutely isn’t the case that men might be able to fight prostate cancer by smoking cannabis,” she added.
Well, well, well, leave it to the MSM to misrepresent the facts and miss the real story. First, the chemicals assessed in this study, R(+)-Methanandamide and JWH-015, are neither “cannabinoids” nor are they “chemicals in cannabis.” Rather, they are synthetic, selective CB2 receptor agonists. In short, they are chemicals created in a lab to mimic certain elements in marijuana, and to bind to specifically to those cannabinoid receptors that are not located in the brain. After all, we can’t possibly have the terminally ill feeling ‘better’, now can we?
Second, US federal researchers have known for some 35 years that the naturally occurring chemicals in cannabis — not just synthesized agonists — can halt the proliferation of multiple types of cancer, including including brain cancer, prostate cancer, breast cancer, lung cancer, skin cancer, and pancreatic cancer. We even know how.
Cannabinoids: potential anticancer agents
via Nature Reviews Cancer (2003)
Cannabinoids are usually well tolerated, and do not produce the generalized toxic effects of conventional chemotherapies. … Cannabinoids inhibit tumor growth in laboratory animals. They do so by modulating key cell-signaling pathways, thereby inducing direct growth arrest and death of tumor cells, as well as by inhibiting tumor angiogenesis and metastasis. Cannabinoids are selective anti-tumor compounds, as they can kill tumor cells without affecting their non-transformed counterparts.
Of course, the real question — conveniently ignored by Reuters and the rest of the MSM — is this: Why, after three decades and dozens of preclinical trials documenting cannabis’ potent anti-cancer abilities, are “many more years of testing” necessary? Last I checked, humans die en masse from cancer, not rats! Yet for some 35 years scientists have been content to replicate these cancer-killer findings in animals and in petri dishes, all the while warning, “It absolutely isn’t the case that men might be able to fight prostate cancer by smoking cannabis.”
Well why the hell not? Not only can cannabis alleviate cancer patients’ nausea and pain, elevate their mood, and increase their appetite, but also — as dozens of preclinical trials over the past three decades consistently demonstrate — marijuana may help to alleviate the very disease that’s ravaging their bodies. Of course, rather than put this theory to the test, investigators for more than three decades have been willing to let people with a terminal illness die while they piddle around with their petri dishes. And to date, not one reporter from the mainstream media has ever had the guts to ask them why.
View Comments
--------------------------------------------------------------------------------
If Cannabis Smoking Didn’t Adversely Impact Lung Function You Would Have Read About It, Right?
Tue, 18 Aug 2009 20:51:23 By: Paul Armentano, NORML Deputy Director
Share This Article
To follow up on yesterday’s blog post, here are the findings of yet another just published study that the mainstream media will undoubtedly ignore.
Effects of cannabis on lung function: a population-based cohort study
via nih.gov
The effects of cannabis on lung function remain unclear and may be different to tobacco. We compared the associations between use of these substances and lung function in a population-based cohort (n=1037). … Cumulative cannabis use was associated with higher forced vital capacity, total lung capacity, functional residual capacity, and residual volume. Cannabis was also associated with higher airways resistance but not with forced expiratory volume in 1 second, forced expiratory ratio, or transfer factor. These findings were similar amongst those who did not smoke tobacco.
By contrast, tobacco use was associated with lower forced expiratory volume in 1 second, lower forced expiratory ratio, lower transfer factor, and higher static lung volumes, but not with airways resistance. Cannabis appears to have different effects on lung function to those of tobacco.
Just in case you think that this is the first time that researchers have failed to document a decline in lung function in marijuana users, well, think again. And again. And again.
